Endothelial dysfunction is one of the important mechanisms of organ and tissue damage in sepsis. In this study, we evaluated the effects of neohesperidin dihydrochalone (NHDC) on lipopolysaccharide (LPS)‐induced vascular dysfunction and explored the potential mechanisms.

Neohesperidin dihydrochalcone (NHDC) reduces lipopolysaccharide-induced vascular dysfunction in sepsis by regulating antioxidant capacity.

Paper

Neohesperidin Dihydrochalcone Alleviates Lipopolysaccharide‐Induced Vascular Endothelium Dysfunction by Regulating Antioxidant Capacity

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